Is there reason to worry about the new Delta variant? Or, for that matter, any other variant? The answer, according to Michael Yeadon, Ph.D., a life science researcher and former vice-president and head scientist of allergy and respiratory research at Pfizer, is a resounding “NO.” Yeadon explains why in the interview above, which is part of the full-length documentary “Planet Lockdown.”
“Basically, everything your government has told you about this virus, everything you need to do to stay safe, is a lie,” Yeadon says. “Every part of it … None of the key themes that you hear talked about — from asymptomatic transmission to top-up vaccines [i.e., booster shots] — not one of those things is supported by the science.
Every piece is cleverly chosen adjacently to something that probably is true, but is itself a lie, and has led people to where we are right now.”
When it comes to your susceptibility to variations, or altered versions of SARS-CoV-2, your resilience is more dependent on T-cell immunity, commonly known as cellular immunity. Yeadon elaborates:
“You’ve got four or five different arms of the immune system: innate immunity, mucosal, antibody, T-cells and compliment[ary systems]. There are all of these different wonderful systems that have integrated, one with another, because it needs to defend you against all sorts of different threats in the environment.
What I’m telling you is that the emphasis on antibodies in respect of respiratory viral infections is wrong, and you can establish that quite easily by doing some searching …
I’m not saying antibodies have no role, but they’re really not very important. This has been proven. There are some people in whom a natural experiment has occurred. They have a defect and they actually don’t make antibodies, but they’re able to fight off COVID-19, the virus SARS-CoV-2, quite well.
The way they do that is, they have T-cell immunity, cellular immunity. [T-cells] are cells that are trained to detect virus-infected cells and to kill those cells.
That’s how you defend yourself against a virus. So, all of these mentions of antibody levels, it’s just bunk. It is not a good measure of whether or not you’re immune. It does give evidence that you’ve been infected, but their persistence is not important as to whether you’ve got immunity …
We’ve known this for decades. We’ve known about T-cells for decades. They were clearly in my undergraduate textbooks. And we’ve known about their importance in defending you against respiratory viruses since probably the 1970s, certainly the 1980s …
It’s quite normal for RNA viruses like SARS-CoV-2, when it replicates, to make typographical errors. It’s got a very good error detection, error correction system so it doesn’t make too many typos, but it does make some, and those are called ‘variants.’
It’s really important to know that if you find the variant that’s most different from the sequence identified in Wuhan, that variance … is only 0.3% different from the original sequence.
I’ll say it another way. If you find the most different variance, it’s 99.7% identical to the original one, and I can assure you … that amount of difference is absolutely NOT possibly able to represent itself to you as a different virus. [So] when your government scientists tell you that a variant that’s 0.3% different from SARS-CoV-2 could masquerade as a new virus and be a threat to your health, you should know, and I’m telling you, they are lying.”
TO SUMMARIZE
Yeadon is stating that a virus cannot mutate into a form that is so distinct to the original that your body cannot recognise it. If you have T cell immunity, your immune system will detect the modified virus and deal with it in the same way as it would with the original virus.
He discusses how, early in the epidemic, investigators collected blood from individuals who had become ill with the SARS virus 17 or 18 years before. SARS-CoV-1, which caused the SARS outbreak, is only 80% identical to SARS-CoV-2. They wanted to see if these patients’ immune systems could detect SARS-CoV-2, which they did.They still maintained memory T-cells against SARS-CoV-1, and those cells identified SARS-CoV-2, despite the fact that they were only 80 percent identical.
Now, if a 20 percent difference was not enough to circumvent the immune system of these patients, why should you be concerned with a variant that is at most 0.3 percent different from the original SARS-CoV-2? And why would we need booster shots for these near-identical variants?
Source : Planet Lockdown
